Reported in some neurons principally when the virus was inoculated Some experimental studies that have been performed, apoptosis were Rabies does not appear to induce massive neuronal loss. Viral particles (Iwasaki & Tobita, 2002). Negri bodies are formed by a granular matrix surrounded by Observed with conventional anatomical and histopathological analysis Presence of eosinophilic cytoplasmic inclusions called Negri bodies are Symptoms, rabies produces only a low inflammatory and microglial Spinal cord and advances to the brain, causing marked neuronalĭysfunction with fatal results. Terminals of the new host, it is transported to the motor neurons of the The rabies virus spreads through retrograde axonal transport from Humans and domestic animals, increasing the risk of disease transmission The destruction of natural habitats and climate change areįactors that cause vector displacement to areas that are inhabited by Still approximately 55.000 deaths reported yearly due to rabies Despite the availability of preventive controls, there are In addition, it is almost impossible to eradicate a virus that has wild The entire population is not vaccinatedīecause of the high cost associated with the vaccine and other factors. Rabies can be prevented by the vaccination of pets and people at high Rabies remains a public health problem in developing countries. APA style: Differential Effect of the Route of Inoculation of Rabies Virus on NeuN Immunoreactivity in the Cerebral Cortex of Mice/ Efecto Diferencial de la Via de Inoculacion del Virus de la Rabia sobre la Inmunorreactividad de NeuN en la Corteza Cerebral de Ratones.Differential Effect of the Route of Inoculation of Rabies Virus on NeuN Immunoreactivity in the Cerebral Cortex of Mice/ Efecto Diferencial de la Via de Inoculacion del Virus de la Rabia sobre la Inmunorreactividad de NeuN en la Corteza Cerebral de Ratones." Retrieved from
2016 Universidad de La Frontera, Facultad de Medicina 17 Oct.
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